Secondary necrotic neutrophils release interleukin-16C and macrophage migration inhibitory factor from stores in the cytosol

S Roth, M Agthe, S Eickhoff, S Möller, CM Karsten… - Cell death …, 2015 - nature.com
S Roth, M Agthe, S Eickhoff, S Möller, CM Karsten, N Borregaard, W Solbach, T Laskay
Cell death discovery, 2015nature.com
Neutrophils harbor a number of preformed effector proteins that allow for immediate
antimicrobial functions without the need for time-consuming de novo synthesis. Evidence
indicates that neutrophils also contain preformed cytokines, including interleukin (IL)-1ra,
CXCL8 and CXCL2. In the search for additional preformed cytokines, a cytokine array
analysis identified IL-16 and macrophage migration inhibitory factor (MIF) as preformed
cytokines in lysates from human primary neutrophils. Both IL-16 and MIF are unconventional …
Abstract
Neutrophils harbor a number of preformed effector proteins that allow for immediate antimicrobial functions without the need for time-consuming de novo synthesis. Evidence indicates that neutrophils also contain preformed cytokines, including interleukin (IL)-1ra, CXCL8 and CXCL2. In the search for additional preformed cytokines, a cytokine array analysis identified IL-16 and macrophage migration inhibitory factor (MIF) as preformed cytokines in lysates from human primary neutrophils. Both IL-16 and MIF are unconventional cytokines because they lack a signal sequence. Using confocal immunofluorescence microscopy as well as western blot analysis of subcellular fractions, IL-16 and MIF were found to be stored in the cytosol rather than in the granules of human neutrophils, which implies an unconventional secretion mechanism for both cytokines. IL-16 is synthesized and stored as a precursor (pre-IL-16). We present evidence that the processing of pre-IL-16 to the biologically active IL-16C is mediated by caspase-3 and occurs during both spontaneous and UV-induced apoptosis of human neutrophils. Although IL-16 processing occurs during apoptosis, IL-16C and MIF release was observed only during secondary necrosis of neutrophils. Screening a panel of microbial substances and proinflammatory cytokines did not identify a stimulus that induced the release of IL-16C and MIF independent of secondary necrosis. The data presented here suggest that IL-16 and MIF are neutrophil-derived inflammatory mediators released under conditions of insufficient clearance of apoptotic neutrophils, as typically occurs at sites of infection and autoimmunity.
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